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Taken together, these observations suggest that except in vitamin B 12 deficiency (which only responds to cyanocobalamin treatment because of the inactivation of methionine synthase), folate treatment should be the most adequate therapy to reverse hyperhomocysteinemia detected both under basal conditions and after an oral methionine load

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Mutations in human CCP1 cause infantile-onset spinocerebellar degeneration, characterized by cerebellar atrophy and motor neuropathy [122]

doi: 10.3390/nu15183953 204

Results showed that a twice daily IGF-1 dose of 120mcg/kg produced almost identical effects as that of a twice daily 80mcg/kg dose [18]